While Traumatic Brain Injuries (TBIs) have been described as the "signature wound" of the current American military, brain injuries have occurred for as long as humans have been on the planet. While some head injuries can be severely disfiguring and produce massive physical and psychological deficits, some TBIs are more subtle and can go undetected and/or untreated. This course discusses and analyzes the effects of more subtle TBI caused by prenatal teratogens (e.g., fetal alcohol syndrome) and postnatal toxic events (e.g., seizures), anchoring these deficits in the basic psychophysiology of the brain. Assessment methods and tools for detecting brain dysfunction are discussed and sample case data are presented to illustrate diagnostic patterns. Lastly, the clinical implications of these injuries are reviewed across several relevant contexts.
This online course is approved for APA CE credit, NBCC CE clock hours, ASWB Clinical CE clock hours, and NYSED CE credit.
- Common Teratogens and Postnatal Processes: Sources of the Silent Slaughter
- Anatomy and Psychophysiology of the Human Brain: A Brief Overview Related to Teratogens and Early Developmental Toxicity
- Assessment Methods for Detecting Brain Dysfunction
- Sample Assessments
- Clinical Implications
After completing this course, health professionals will be able to:
- Define teratogen and identify 7 common prenatal and postnatal trauma sources that are associated with traumatic brain injury (TBI) of a more subtle nature.
- Discuss the basic psychophysiology of the brain as it relates to teratogens, postnatal toxic events, and TBI.
- Describe common assessment methods in this context, including the clinical interview, 5 standardized psychological instruments, and 5 neurological tests to assess brain dysfunction.
- Utilize test results presented from 8 diverse case samples to guide in their ability to clinically detect and treat these subtle TBIs.